Anti-Amoebic Drugs

Anti-Amoebic
Drugs

Contents

       Antiamoebic Agents – Classification

       Pharmacology of Metronidazole

At the
end of this lecture, the student will be able to:

       Describe
the Pharmacology of Metronidazole

       Explain
the ADR and drug interactions of Metronidazole

Introduction

       Amoebiasis
(Amoebic dysentry) is an infectious disease caused by protozoa, Entamoeba
histolytica,
which is produced by the ingestion of cysts of this organism

       The
ingested cysts develop into trophozoites and adhere to the colonial epithelial
cells in the intestine

       These
trophozoites then lyses the host cell and invades the submucosa

       This
produces the amoebic ulcers and cause acute dysentery or chronic intestinal
amoebiasis

       The
parasite may also pass into blood steam and invades the liver causing liver
abscesses

Classification

1. Systemic Amoebicides

     a) For both
intestinal and extra intestinal amoebiasis

     
Nitromidazoles: Metronidazole, Tinidazole, Secnidazole, Ornidazole

      – Alkaloids:
Emetine, Dehydroemetine

     b) For extra intestinal
amoebiasis: Chloroquine

2. Luminal Amoebicides

     a) Amides:
Diloxanide furoate

     b)
8-Hydroxyquinolines: Diiodohydroxyquin

     c) Antibiotics:
Tetracyclines, Paromomycin

Metronidazole

       Metronidazole
is the drug of choice in the treatment of different forms of amoebiasis

       It
kills the trophozites of E. histolytica but has no effects on the cysts

       It
is often used in combination with Diloxanide furoate for the treatment of
amoebiasis

Mechanism of Action

Metronidazole is a pro durg.

It is converted in anaerobic organism by the redox enzyme
pyruvate-ferredoxin oxidoreductase. The notro group of metronidazole is
chemically reduced by ferredoxin or a ferredoxin linked metabolic process and
the products are responsible for disrupting the DNA helical structure, thus
inhibiting nucleic acid synthesis.

Pharmacokinetics

       Disposition
of metronidazole in the body is similar for both oral and intravenous dosage
forms

       Following
oral administration, metronidazole is well absorbed

       Plasma
concentrations of metronidazole are proportional to the administered dose

       Metronidazole
is the major component appearing in the plasma, with lesser quantities of
metabolites also being present

       Less
than 20% of the circulating metronidazole is bound to plasma proteins

       Metronidazole
appears in cerebrospinal fluid, saliva, and breast milk in concentrations
similar to those found in plasma

       Bactericidal
concentrations of metronidazole have also been detected in pus from hepatic
abscesses.

       The
major route of elimination of metronidazole and its metabolites is via the
urine (60% to 80% of the dose)

Anti microbial spectrum

       Metronidazole
has a broad-spectrum cidal activity against protozoa and many anaerobic
bacteria

       It
is drug of choice in treatment of infections caused by E. histolytica, Giardia
lamblia
and Trichomonas vaginalis

       It
is also active against Gram-positive bacilli such as Clostridia

Adverse Effects

       The most common unwanted effects are
gastrointestinal disturbances

       It has a metallic, bitter taste in
the mouth

       CNS symptoms such as dizziness,
headache and sensory neuropathies are rarely observed

       If taken with alcohol a disulfiram
like effect occurs

Contraindications

Metronidazole
is contraindicated in:

       Neurological disease

       Blood dyscrasias

       First trimester of pregnancy (though
no teratogenic effect has yet been demonstrated, its mutagenic potential
warrants caution)

       Cautious use in chronic alcoholics.

Interactions

       Disulfiram-like intolerance to
alcohol occurs in some patients taking metronidazole

       Alcohol-metronidazole interaction
occurs only in some individuals, while majority of those taking it can consume
alcohol without any reaction

       There is no convincing evidence of
disulfiram-like action of metronidazole, but manufactures advise caution in
drinking during metronidazole therapy

       Enzyme inducers (phenobarbitone,
rifampin) may reduce its therapeutic effect

       Cimetidine + metronidazole can
reduce metronidazole metabolism: its dose may need to be decreased

       Metronidazole enhances warfarin
action by inhibiting its metabolism

       It can decrease renal elimination of
lithium and precipitate toxicity

Clinical Uses

       It is used in the treatment of
amoebiasis, giardiasis, trichomonas vaginitis and pseudomembranous enterocolitis

       Also used in the treatment of many
anaerobic bacterial infections and in peptic ulcers

Summary

       Amoebiasis
(Amoebic dysentry) is an infectious disease caused by protozoa, Entamoeba
histolytica,
which is produced by the ingestion of cysts of this organism

       The
nitro group of metronidazole is chemically reduced by ferrodoxin and the
product of the reaction disrupts nucleic acid synthesis

       ADR
of metronidazole- causes
metallic, bitter taste in the mouth; dizziness, headache and sensory
neuropathies are rarely observed; causes a disulfiram like effect
with
alcohol

 

 

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